Abstract
Myosin VI is a multi-functional cytoskeletal motor which acts as a transporter during endocytosis and as an anchor to maintain stereocilia structure. Malfunctions in motor enabled processes are implicated in pathologies including cancer and deafness; thus, mechanisms through which myosin VI cellular roles are regulated are of importance. Recruitment of myosin VI on the cargo is enabled by adapter proteins such as GIPC and Dab2 that impart structural and mechano-chemical changes in the motor, making the motor more versatile and useful for multiple intracellular processes.
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