Abstract
To the Editor: With great interest we read the article by Malavazos et al1 discussing the possible pathophysiological role for cardiotrophin-1 (CT-1) in left ventricular growth. CT-1 is a member of the interleukin-6 superfamily, which induces cardiomyocyte growth. CT-1 provides prognostic information in patients with untreated essential hypertension and associates with the magnitude of left ventricular hypertrophy in these patients.2 In contrast, plasma CT-1 is decreased in hypertensive rats.3 Recently, Natal et al4 identified adipose tissue as a source of CT-1. They found an elevated level …
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