Abstract
Cardiac troponin (cTn) is an intracellular protein that is expressed by normal myocardial cells. Myocardial cell injuries that are caused by severe myocardial ischemia can lead to the persistent release of cTn, which results in a sustained elevation in the peripheral circulation that can last approximately 5–7days for cTn I and 7–14days for cTn T [1]. During this process, many types of cells, including myocardial cells, are exposed to high concentrations of cTn in the extracellular fluid. However, it is unknown whether a sustained high concentration of cTn has direct biological effects on adjacent myocardial cells during serious ischemic injury.
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