Abstract
Low epidermal filaggrin (FLG) is a major risk factor for the development and progression of atopic dermatitis (AD) and atopic march sequelae. We recently found that low non-lesional, but not lesional, FLG levels are associated with the development of co-sensitization and food allergy. We hypothesized that variance in genetic loci other than FLG contribute to low epidermal FLG expression in non-lesional skin.
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