Abstract
Thin filaments isolated from chicken gizzard smooth muscle in either the presence or the absence of Ca 2+ possess identical caldesmon contents. Hence, a ‘flip-flop’ mechanism, involving Ca 2+-dependent association and dissociation of caldesmon and thin filaments, does not appear to operate in vivo and is an unlikely model for caldesmon function.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callMore From: Biochimica et Biophysica Acta (BBA) - Molecular Cell Research
Disclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
