Abstract
SPS is most likely a hereditary, autosomal dominant thrombophilia characterized by platelet hyperaggregation after low concentrations of platelet inducers - adenosine diphosphate (ADP) and/or epinephrine (EPI). It manifests with angina pectoris, acute myocardial infarction (MI), transient cerebral ischemic attacks (TIA), stroke, retinal thrombosis, early pregnancy loss syndrome, peripheral arterial thrombosis, and venous thrombosis, sometimes recurrent under oral anticoagulant therapy.
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