Abstract
Purpose: C-reactive protein (CRP) is an independent marker for inflammation and cardiovascular disease. In addition, it has many biological functions which may affect atherogenesis and vascular disease formation; however, the underlying molecular mechanisms are largely unknown. Accumulation of lipoprotein-derived cholesterol inside macrophages is one of crucial mechanisms of atherogenesis. The aim of this study was to investigate the effects and molecular mechanisms of CRP on cholesterol extracellular transport (cholesterol efflux) from human macrophage-derived foam cells.
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