Abstract
AbstractThe compensatory response is a regulatory event influencing the redundancy of the ribosomal RNA cistrons (rDNAs) of Drosophila melanogaster. In this report we attempt to demonstrate that the compensatory event and the thymidine analogue bromodeoxyuridine (BrdU) specifically interact. We conclude that the drug inhibits the compensatory response of Drosophila melanogaster XO males and argue that the compensatory event is not the passive consequence of replicational dominance known to occur in Drosophila polytene tissues.
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