Abstract

Objectives: Find and eradicate the root cause/s of Primary & Secondary Hypertension.For a long time, Syndrome X has been regarded as a root cause of primary & secondary Hypertension. The prevailing cause of this ghost syndrome has never been documented, defined or eradicated as yet. However, the patients worldwide are being maintained with a variety of Neuro-electrical (Voltage & Charge) & Vascular (Hemodynamic) manipulative medicine and radically performing the Renal Sympathetic Ablation, to sustain a normal blood pressure. Great many possible parameters have been established to view the existence of the hypertension, Bacterial, Viral Hormonal and anatomical. In the recent years, scientists are looking closely at the Neuroinvasive viruses and their role in the cardiovascular disease. Most scientists agree to a common instigator of the cardiac pathology that anatomically leads to hypertension. The pathological issues can only be resolved by the medical intervention. All conventional treatments provide the symptomatic resolution only. Very little work has been done on the understanding of the Neuro-cardiac pathology induced by herpes family of viruses. The electrical voltage transitional activity can be seen changed, under the influence of the electrolyte imbalance of the myocardium cells i.e. high/ Low Na+ or high/ Low Potassium or Calcium. The pathogenesis of primary hypertension is multifactorial. However, the sympathetic nervous system plays an important role in circulatory and metabolic control and has clearly been established as a major contributor to the development of hypertension, with blood pressure elevation being initiated and sustained by elevated sympathetic nervous activity. Increased sympathetic outflow to the heart resulting in increased cardiac output and neurally mediated vasoconstriction of peripheral blood vessels are obvious examples of neural pathophysiological pathways leading to elevated blood pressure. The consequences of increased sympathetic outflow to the kidneys, perhaps most important in this context are sodium and water retention, increased renin release and alterations of renal blood flow, effects that contribute substantially to blood pressure elevations both acutely and in the long term. Accordingly, targeting the sympathetic nervous system directly appears to be a logical therapeutic approach for the treatment of hypertension. However, Sympathetic nervous system is not immune against the viral infection susceptibility. Therefore, we have studied pathological issues driven by the Neuroinvasive viruses that give rise to the anatomical and physiological dysfunctions. That collectively results into the hypertension and other cardiac dysfunctions.

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