Abstract
Midpontile ischemic decerebration in vagotomized mesencephalic cats produced marked tachycardia and hypertension. Subsequent transection of the brain stem at or above the midpontile level did not affect the cardiovascular hyperactivity. Transection at the pontomedullary junction abolished the brain stem augmentation of heart rate and blood pressure. The results suggest the existence of a cardiovascular augmenter area in the caudal half of the pons and a cardiovascular depressor area in a more rostral portion of the brain stem.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
