Abstract
Blunt-head trauma is a major cause of disability giving rise to neurological deficits, neuropsychological deficits, and epilepsy. Necropsy studies have identified surface contusion and lacerations, diffuse axonal injury, affecting predominantly central myelinated structures, and ischaemia as main mechanisms of damage.1 To localise damaged regions and measure the damage in vivo we used diffusion-tensor imaging (DTI). We investigated a 42-year-old man who had suffered from a severe blunt-head trauma in a road traffic accident with skull fracture and right frontotemporal extradural haemorrhage 9 years ago.
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