Abstract
We investigated the ability of various blood markers to detect an alcoholic cause of acute pancreatis. Serum carbohydrate-deficient transferrin (CDT) was significantly correlated with reported 2 month and 7 day ethanol consumptions and was significantly higher in 42 patients with alcoholic acute pancreatitis and in 24 patients with possibly alcoholic acute pancreatitis than in 20 patients with non-alcoholic disease. At a cutoff over 17 U/L, the specificity of CDT was 100% and the sensivity was 75% to detect an alcoholic cause of acute pancreatitis. —The lipase/amylaseratio index, erythrocyte mean corpuscular volume, and gamma glutamyl transferase could not distinguish alcoholic from non-alcoholic acute pancreatitis. Lancet 1994; 343: 1328–29
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