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Abstract
Objectives: Heart failure (HF) is often the consequence of damage to the myocardium during which remodelling and de-differentiation of cardiomyocytes is observed. During the course of the disease the infiltration of the myocardium with inflammatory cells is well recognized and becomes nowadays a major focus of research. We hypothesized that oncostatin M (OSM), a cytokine mainly restricted in its expression to infiltrating macrophages, is a major contributor to heart failure. In addition, the exceptionally high level of the OSM receptor-β (Oβ) on cardiomyocytes stimulated our work.
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