Abstract
Perinatal exposures to Bisphenol A (BPA) have been associated with an increase in asthma prevalence. We have reported the increased expression of bromodomain-containing protein 4 (BRD4), zinc finger DHHC-1, and stimulators of interferon genes (STING) in BPA exposed mouse immune cells. The purpose of this study was to test the hypothesis that perinatal exposure to BPA enhances allergic asthma development through BRD4/ ZDHHC-1/STING axis.
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