Abstract
This article presents a clinical case of drug-induced acute TIN that developed in response to self-administered intake of bismuth tripotassium dicitrate. This clinical case was characterized by an acute onset, presence of urinary syndrome (low specific gravity of the urine and proteinuria), occurrence of azotemia in the setting of preserved diuresis, lack of hyperkaliemia, and quick and complete restoration of renal functioning after the discontinuation of a bismuth-containing drug. It can be suggested that the presented clinical case will allow for a better understanding of the causes and clinical features of acute TIN and the choice of the management tactics for patients with this pathology.
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