Abstract
Purpose: Osteoarthritis (OA) is the most common rheumatological disease and a major cause of pain and disability in older adults. Currently no treatment is available that alters the course of OA. Mitochondrial dysfunction in chondrocytes is associated with the molecular dysregulation underlying OA, and has been proposed as a potential therapeutic target. APPA, a combination of apocynin (AP) and paeonol (PA), has the potential capacity to protect mitochondria from injury. Aim To study the biological effect of APPA in human articular chondrocytes.
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