Abstract
Annals of Reviews & Research open access journal that publishes high-quality solicited and unsolicited articles, from various types of subject areas ranging from management to engineering sciences, accounting, criminal sciences, etc. which deal with innovative research work.
Highlights
A precipitating cause, such as pneumonia, shock, aspiration of stomach contents, sepsis, or trauma, is invariably present in acute respiratory distress syndrome (ARDS)
“lung-protective ventilation” in mechanically ventilated patients with ARDS is the best practice, with no specific therapy aimed at lung inflammation
A complex network of proinflammatory signaling pathways and oxidative stress created by a range of cell types in the lungs initiate, amplify, and control the inflammatory response in patients with ARDS
Summary
A precipitating cause, such as pneumonia, shock, aspiration of stomach contents, sepsis, or trauma, is invariably present in acute respiratory distress syndrome (ARDS). Because of comorbidities such as sepsis, multiorgan failure, refractory shock, and refractory hypoxemia, patients with ARDS have a significant death rate (50 percent) [1]. TLR4 activation causes the generation of pro-inflammatory cytokines, which can increase the severity of injuries, as previously stated. TLR4 is a pattern recognition receptor from the TLR protein family that activates NF-B and causes the production of inflammatory cytokines and chemokines including TNF- and IL-6 in lung cells. Woman’s tongue, Tibet lebbeck, singer-tree, shack-shack, Lebbeck, Lebbek Tree, Flea Tree, Frywood, Koko Shirish, Saras Khok Siridam, Vagai
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