Abstract
Voltage-gated sodium channels are responsible for initiating and propagating action potentials in excitable cells and have been associated with numerous cardiac and neurological disorders. However, there are many channel subtypes with different roles making it a challenging task to develop therapeutics specific to certain subtypes. NaV1.7 channels, present at the endings of pain-sensing nerves, have garnered a lot of scientific interest as potential targets for pain therapeutics by channel inhibition.
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