Abstract
A1 astrocyte exerts neurotoxic effects and is a key factor of failure in nerve repair and myelin regeneration. Bilobalide (BB) is one of the active monomer component of G. biloba extract, which alleviates the demyelination in Cuprizone (CPZ) model and reduces the secretion of inflammatory cytokines by astrocyte. Our aim was to explore possible mechanisms of BB targeting astrocyte.
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