Abstract
Cryptorchidism is a known cause of testicular tumor. The incidence of testicular tumor is 11 times more in inguinal testes and 50 times more in intra-abdominal testes. The position of the undescended testis is related to the likelihood of carcinogenesis with the intra-abdominal location having the highest risk for malignancy. Bilateral testicular tumors are rare and 80% of bilateral tumors are metachronous. Synchronous bilateral testicular tumors are rare and bilateral synchronous testicular seminoma in a patient with bilateral cryptorchidism is very rare.
Highlights
The incidence of testicular germ cell tumor is 0.005% in general population and it incurs 1-5% increased risk of developing germ cell tumor in contralateral testis.[1]
The position of the undescended testis is related to the likelihood of carcinogenesis with the intra-abdominal location having the highest risk for malignancy.[3]
Bilateral synchronus testicular tumor in bilateral cryptorchidism is very rare,[1] and to our knowledge this is the second case being reported in world literature
Summary
The incidence of testicular germ cell tumor is 0.005% in general population and it incurs 1-5% increased risk of developing germ cell tumor in contralateral testis.[1]. Patient revealed on enquiry that both the testes were absent in scrotum since his birth He had been married for 15 years with a history of normal conjugal life but did not have any children. Radiological impression was bilateral testicular tumor with differential diagnosis (DD) of Non Hodgkin’s Lymphoma (NHL) and mesenteric fibromatosis (desmoid tumor) On gross examination, both tumors showed similar grey white cut surface without any necrosis, hemorrhage and cystic change (Figure 3a,3b). With this history bilateral testicular tumor was considered as the first DD and serum enzyme assays were done. The serum α-fetoprotein (AFP) level was 9.2 ng/ml, serum beta-human chorionic gonadotrophin (β-hCG) was 1.1 ng/ml and serum lactate dehydrogenase (LDH) was marginally elevated (519 U/L)
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