Bilateral injury of deep peroneal nerve in the patient after heart transplant

Adam Śmiechowski,Małgorzata Sobieszczańska-Małek

doi:10.15829/1560-4071-2020-3790

Abstract

In March 2015, a 62-year-old patient with advanced heart failure underwent a failed radiofrequency ablation, followed by paresis of muscles in the anterior compartment of the leg. After rehabilitation, partial recovery of the paresis was achieved. Orthotopic heart transplantation was performed 9 months after ablation at the at the Institute of Cardiology, followed by a bilateral paresis of muscles in the anterior leg compartment. Rehabilitation was implemented. The possible cause of paresis is most likely to be due to food shortages, mainly related to a folic acid deficiency.

Highlights

Clinical situation In September 2015, a 62-year-old patient with dilated cardiomyopathy, NYHA class III heart failure (HF), left ventricular ejection fraction (LVEF) of 44%, severe mitral regurgitation, history of asystole in 2012, followed by implantation of a cardioverter-defibrillator was admitted to the National Institute of Cardiology
On December 23, 2015, a heart transplant was per formed at the National Institute of Cardiology
Neurologist supposed that a possible cause of palsy is fibular nerve compression at the fibula head

Summary

КЛИНИЧЕСКИЙ СЛУЧАЙ

Bilateral injury of deep peroneal nerve in the patient after heart transplant Adam Śmiechowski, Małgorzata Sobieszczańska-Małek. In March 2015, a 62-year-old patient with advanced heart failure underwent a failed radiofrequency ablation, followed by paresis of muscles in the anterior compartment of the leg. Orthotopic heart transplantation was performed 9 months after ablation at the at the Institute of Cardiology, followed by a bilateral paresis of muscles in the anterior leg compartment. Bilateral injury of deep peroneal nerve in the patient after heart transplant. Двустороннее поражение глубокой ветви малоберцового нерва у пациента после трансплантации сердца. Clinical situation In September 2015, a 62-year-old patient with dilated cardiomyopathy, NYHA class III heart failure (HF), left ventricular ejection fraction (LVEF) of 44%, severe mitral regurgitation, history of asystole in 2012, followed by implantation of a cardioverter-defibrillator was admitted to the National Institute of Cardiology. The patient’s gait had typical features of fibular nerve palsy, which was

Discussion

Conclusion