Abstract

Background:Clinically significant spontaneous bilateral iliopsoas hematoma is a rare complication of anticoagulation therapy. Definitive treatment of spontaneous iliopsoas hematomas is not well-established and varies between observation and surgical intervention. The intramuscular hematoma causes severe pain, muscle dysfunction, and occasionally nerve palsy with the femoral nerve most commonly affected. Most patients are neurologically normal but when a significant neurological deficit is associated with iliopsoas hematoma, optimal treatment recommendations vary. We report a case of spontaneous bilateral iliopsoas hematomas causing significant bilateral femoral nerve dysfunction.Case Description:The authors present the case of a 63-year-old female who developed bilateral femoral nerve palsy due to anticoagulation bleeding complication. Magnetic resonance imaging demonstrated large bilateral intramuscular psoas hematomas causing femoral nerve compression. Surgical evacuation and decompression of the femoral nerves was performed with rapid neurological improvement.Conclusion:Management recommendations depend on the volume and cause of the hematoma, timing of diagnosis, and the degree of neurological impairment. A conservative approach with bed rest and correction of bleeding abnormalities to allow the hematoma to spontaneously resorb has been utilized for patients with small hematomas and little to no neurological symptoms. In contrast, more aggressive recommendations have been made for patients with large hematomas, severe motor function deficits, or hemodynamic instability.

Highlights

  • Significant spontaneous bilateral iliopsoas hematoma is a rare complication of anticoagulation therapy

  • Most patients are neurologically normal but when a significant neurological deficit is associated with iliopsoas hematoma, optimal treatment recommendations vary

  • We report a case of spontaneous bilateral iliopsoas hematomas causing significant bilateral femoral nerve dysfunction

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Summary

Conclusion

Management recommendations depend on the volume and cause of the hematoma, timing of diagnosis, and the degree of neurological impairment. The patient was in no apparent distress and stable hemodynamically She had normal tone, but moderate bilateral proximal lower extremity weakness with 3/5 strength in bilateral iliopsoas and 2/5 strength in quadriceps. On day 11 of hospitalization, it was noted that the patient’s strength had deteriorated further with iliopsoas and quadriceps strength of 1/5, bilaterally Based on this neurological deterioration, she was taken for surgical decompression of the hematomas through bilateral flank incisions. By the third postoperative day, the patient demonstrated marked improvement in strength and near complete resolution of her thigh numbness; both iliopsoas muscles had 4/5 power. Iliopsoas strength remained 4/5 bilaterally at 6‐week follow‐up Her left quadriceps improved to 5/5 and her right quadriceps improved to 1/5. At 3‐ and 6‐month follow‐up visits, the patient was noted to have full strength in her lower extremities, but experienced continued mild dysesthesias in anterolateral thighs bilaterally

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