Abstract
By what mechanism do clusters of transcription factors–in constant and rapid protein subunit exchange with their environment–hierarchically assemble and regulate transcription? How do clusters, such as clusters of T-cell receptors or estrogen receptors, differ in assembly kinetics and composition across cell membranes? Can we follow the reaction kinetics of interacting pairs of biomolecules in an effort to probe their heterogeneity, such as the human linker histone H1 and histone chaperone prothymosin α?
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