Abstract
Patients with heart failure and preserved ejection fraction (HFpEF) are supersensitive to volume overload, and flash pulmonary edema often occurs transiently which is rapidly resolved by intravascular volume reduction. The stressed blood volume and systemic blood pressure are controlled by several systems. Among them, the baroreflex system is an important and powerful regulator. Major risk factors of HFpEF also promote atherosclerosis and thereby baroreflex failure. Therefore, we hypothesized that baroreflex failure plays a pivotal role in the pathogenesis of HFpEF independent of left ventricular (LV) dysfunction.
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