Abstract
Background: Myasthenia gravis (MG) is an antibody mediated disease in which the target autoantigen is the acetylcholine receptor (AChR) at the postsynaptic membrane of the neuromuscular junction. B cell activating factor (BAFF) is the potent B cell survival factor and is necessary for peripheral B cell differentiation and maturation. Excess BAFF promotes the survival and growth of autoreactive B cells. The previous studies have shown that serum BAFF levels in patients with MG are higher than in control subjects. BAFF may play an important role in the pathogenesis of MG.
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