Abstract
The bacteriology of cholecystitis, as worked out heretofore, is largely the bacteriology of the contents and not the tissue of the gall-bladder wall. (The mechanism of infection of the gall-bladder and the production of gall-stones have not been fully worked out experimentally.) In a previous paper I pointed out that when streptococci, from various sources, attain a certain grade of virulence, they are prone to produce on intravenous injection cholecystitis and gall-stones, with large numbers of streptococci as the nuclei. In this connection the observations in the following case are of interest: A woman, aged 62, with infected tonsils, high blood-pressure, and a chronic arthritis, involving chiefly the small joints of the hands, contracted a "cold" and acute tonsillitis. Ten days later she developed a typical acute attack of cholecystitis. The symptoms continued for three weeks without jaundice. Then the pain shifted and the next day jaundice appeared, which deepened
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