Abstract
Abstract Lesions in the septal area and amygdaloid complex impaired the sudomotor, but not the cardiac component of the orienting response of rats to clicks of different intensities. While the change in heart rate resembled the pattern of slight deceleration observed in control rats, the galvanic skin response of brain-damaged animals was markedly decreased. This pattern of autonomic responsivity in brain-damaged animals is of interest in the light of reports of somatic hyperreactivity in septal animals and in the light of amygdalectomized monkeys' behavioral deficits in habituation. Two hypotheses were tentatively entertained in explanation of the data. The one idea considered GSR as an indication that events are encoded in the central nervous system. The other regarded GSR in terms of wider autonomic implications, as an indication of loss of sympathetic tonus.
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