Abstract
Neural control of resting pulse rate was studied in 12 subjects during high spinal anesthesia alone and during high spinal anesthesia plus intravenous atropine. Spinal anesthesia was used to produce complete preganglionic sympathetic block because of its freedom from pharmacologic side effects on heart rate. Sympathetic block slowed the resting heart rate; atropine elevated it to rates above basal control levels. The data confirm that the resting heart rate is influenced in man by both sympathetic and parasympathetic influences but that the latter predominate. Atropine elevates arterial blood pressure in the presence of complete sympathetic block.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
