Abstract
Idiopathic inflammatory myopathies (IIM) involve chronic inflammation of skeletal muscle and subsequent muscle degeneration due to an uncontrolled autoimmune response. The mechanisms leading to pathogenesis are not well understood, however IIM may be triggered by increased exposure of intramuscular antigens due to the defective sarcolemmal membrane repair. Sarcolemmal membrane repair involves an active resealing mechanism that restores barrier function of the membrane after injury and is a critical component of maintaining normal cellular physiology.
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