Abstract
Hearts of rats with diabetes mellitus are generally characterized by energy demands exceeding their energy production. Nevertheless, although working permanently in energy deficiency, the diabetic hearts may also exhibit decreased vulnerabil-ity to ischemia and calcium overload. This points to presence of adaptation changes in cardiac energetics, in which at least that limited amount of energy which the diabetic heart mitochondria can produce, may be transported through the mitochondrial membrane to sites of its utilization at a non-limiting rate.
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