Abstract
Introduction: Tumor-induced damage in the cochlear nucleus may be responsible for the limited success of the auditory brainstem implant (ABI) in neurofibromatosis type 2 (NF2) patients. Therefore, an alternative implantation site that bypasses the damaged pathways in the brainstem may provide better hearing performance in NF2 patients. The central nucleus of the inferior colliculus (ICC) provides access to almost all ascending auditory information and has a well-defined tonotopic organization, which is important for an auditory prosthesis. The auditory midbrain implant (AMI) is a single-shank multielectrode array designed according to the dimensions of the human ICC with the goal of stimulating the different frequency layers of its central nucleus.
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