Abstract

AbstractA 4‐year‐old, neutered, female Jack Russell terrier cross was presented for an acute onset of obtundation, severe hypoglycaemia and generalised tonic‐clonic seizure activity. Baseline cortisol and subsequent adrenocorticotropic hormone stimulation testing confirmed hypoadrenocorticism, which was atypical as there were no electrolyte abnormalities present during hospitalisation or in the 6‐month follow‐up period. The dog responded to glucocorticoid monotherapy. To our knowledge, this is the only confirmed case of atypical hypoadrenocorticism presenting for hypoglycaemic seizures and bilateral cortical blindness in a dog. Hypoadrenocorticism (typical or otherwise) should be considered in dogs presenting for hypoglycaemic seizures.

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