Abstract
Adenosine-triphosphate (ATP) was determined in the cortex of canine kidneys undergoing hypothermic storage in vitro for 24–72 hours. After a rapid initial decrease during normathermia, continuous hypothermic perfusion resulted in partial restoration of the ATP level. Hypothermic storage with only initial perfusion resulted in a much slower decrease in ATP than did normothermia. The rate of disappearance was slower for an intracellular than an extracellular type of electrolyte solution. The efficacy of the various preservation methods as reflected in the results of functional studies, seems to be correlated with the ATP level in the renal cortex.
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