Abstract
Atopic dermatitis represents the most frequent chronic inflammatory skin disease, affecting up to 7% of adults and 25% of children.1 The pathogenesis includes inflammation, skin barrier dysfunction, dysbiosis, and modulation by environmental and genetic factors. Patients generally present with erythema, xerosis, excoriations, lichenification, and pruritus. It affects the quality of life of both patients and family members, and patients with atopic dermatitis are at higher risk for allergic comorbidities.
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