Abstract
Background: Gap junctional intercellular communication (GJIC) plays important roles in both the normal brain function and pathogenesis of various neurological diseases including Parkinson’s disease (PD). Connexin (Cx) 30 is a major gap junctional protein of astroglia. Previous studies have shown altered Cx30 expression in response to dopaminergic (DA) neuronal damage using various animal models, but the precise roles of Cx30 in DA neuron damage remains unknown.
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