Abstract
1992). Elevated levels ofADMA capable of inhibiting endothelial nitric oxide syn-thase (eNOS) have been found in several disorders includ-ing coronary artery disease (CAD) (Boger 2004). Nowa-days, ADMA is regarded as a novel cardiovascular riskfactor and is degraded mainly by an intracellular enzymetermed dimethylarginine dimethylaminohydrolase (DDAH),after uptake from the circulation. DDAH degrades ADMA tocitrulline and dimethylamine (Dayoub
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