Abstract
The pathogenesis of normal pressure hydrocephalus (NPH) is not fully understood and the relationship between inflammatory reaction extension and the ventricular enlargement is unknown.
Highlights
The pathogenesis of normal pressure hydrocephalus (NPH) is not fully understood and the relationship between inflammatory reaction extension and the ventricular enlargement is unknown
Bilateral subarachnoidal injection of kaolin was administered in the cranial convexities of 20 adult rats
Radiological kaolin extension was defined by the number of kaolin locations showed in the MRI studies
Summary
Introduction The pathogenesis of normal pressure hydrocephalus (NPH) is not fully understood and the relationship between inflammatory reaction extension and the ventricular enlargement is unknown. Methods Bilateral subarachnoidal injection of kaolin was administered in the cranial convexities of 20 adult rats. MRI was obtained using a Bruker Biospec 11.7 T MRI scanner at 14, 60, 90 and 120 days post kaolin injection.
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