Abstract
Voltage-gated sodium channels in sensory neurons have been found to play a crucial role in several chronic painful neuropathies of different aethiology. Pharmacons that exhibit a use-dependent blockade of these channels, like anticonvulsants, antiarrhythmics and local anesthetics, have been proven to be useful drugs in the treatment of chronic neuropathic or inflammatory pain states, epilepsy, migraine and neurodegeneration related to ischaemia.
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