Abstract
Background and Aim: Recent clinical data have shown that obstructive sleep apnea (OSA), through its hypoxia related consequences leads to tissue hypoxia, thereby resulting in oxidative stress, inflammation, and sympathetic system activation, and could therefore be an independent risk factor for non-alcoholic fatty liver disease. The primary aim of this observational study is to study the effect of Obstructive sleep apnoea on the degree of liver stiffness, estimated using transient elastography, a novel technique to assess liver fibrosis.
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