Abstract

Introduction: Elevated intracranial pressure may be due to mass effect but also obstruction of CSF outflow. This can be at the level of the Pacchionian granulations, the presumed mechanism in increased intracranial hypertension, but also due to impaired cerebral venous outflow. Multiple mechanisms have been adopted to lower intracranial pressure which has been recognized as a potential cause of irreversible optic nerve dysfunction. One of the most recent developments has been the advent of venous stenting for presumed venous outflow pathology. This may play a role in patients with idiopathic intracranial hypertension or structural venous abnormalities.

Full Text
Published version (Free)

Talk to us

Join us for a 30 min session where you can share your feedback and ask us any queries you have

Schedule a call