As for blood vessels, the answer regarding lymphatics is often NO.

Abstract

Our knowledge on lymphatic vessel function is lagging that of blood vessels. Baranwal et al.1 help bridge this gap in the present issue of Acta Physiologica, by exploring caveolae in lymphatic function. As models, they use mice constitutively lacking caveolin-1 (Cav-1), the primary protein in caveolae required for their formation in endothelial cells and lymphatic endothelial cell (LEC) with specific caveolin-1 gene deletion. In mammals, there are two circulatory systems, the blood vessels that form a closed circulatory system and the lymphatic vessels, where the latter constitutes a one-way conduit returning filtered interstitial fluid and leukocytes via lymph nodes back to the blood circulation. The lymphatic system thus contributes to fluid balance preservation, absorption of digested fat via intestinal lymphatics and immunosurveillance. Formation of lymph starts as interstitial fluid enters initial lymphatics via paracellular transport facilitated by overlapping button-like junctions that allow passive entry of fluid and macromolecules2 . From its origin lymph is transported centrally via collecting vessels having unidirectional valves and smooth muscle with intrinsic contractility, thus, facilitating unidirectional transport3 .