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Abstract
Endometriosis is a chronic gynecologic disease affecting 10% of reproductive-aged women. While medical therapy and surgery are the cornerstones of treatment, each has associated failure rates. We have shown that non-responders (NRs) to progestins have decreased progesterone receptor (PR) expression; surprisingly estrogen receptors (ERs) alpha and beta were often decreased as well. The decreases in ERs and PR levels in endometriosis may be analogous to receptor negative breast cancer, suggesting other non-hormonal mechanisms are involved in disease progression.
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