Abstract
In the acute phase of CCl(4) poisoning of the rat, we observe a marked increase of arginase activity in the plasma, a contemporaneous significant increase of ornithine, and the disappearance of arginine. Experiments carried out in vivo and in vitro show that the changes in plasma concentration of arginine and ornithine are related to the increase of plasma arginase activity. In the experimental necrotic-type liver damage studied, arginase - even on the plasma level - carries out its activity of transforming arginine into ornithine and urea.
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