Abstract
Eating behavior is constantly being shaped by a convergence of homeostatic demands and motivational parameters set to insure adequate behavioral patterns with a variety of metabolic consequences that send signals to brain structures to the brain structures responsible for the homeostatic parameters. Out of the scores of themes concerning eating behavior, the 18 papers comprising the current Research Topic, “The two-way link between eating behavior and brain metabolism,” happened to be distributed among the three large overlapping fields: reward, appetite, and obesity.
Highlights
Eating behavior is constantly being shaped by a convergence of homeostatic demands and motivational parameters set to insure adequate behavioral patterns with a variety of metabolic consequences that send signals to brain structures to the brain structures responsible for the homeostatic parameters
Blum et al (2014) looked at obesity as a result of over-consumption due to addictive processes and impaired reward functioning. They discuss the relevancy of the Reward Deficiency Syndrome: a genetic/epigenetic phenomenon resulting in a dysfunction of the dopaminergic system intertwined with the system of carbohydrate homeostasis
The rewarding nature of food mediated via dopaminergic system can be enhanced under the condition of excessive stress triggering a vicious circle and leading to maladaptive responses including overconsumption of palatable food and obesity (Sominsky and Spencer, 2014)
Summary
Eating behavior is constantly being shaped by a convergence of homeostatic demands and motivational parameters set to insure adequate behavioral patterns with a variety of metabolic consequences that send signals to brain structures to the brain structures responsible for the homeostatic parameters. Understanding eating behavior is impossible without understanding the mechanisms of reward. Blum et al (2014) looked at obesity as a result of over-consumption due to addictive processes and impaired reward functioning.
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