Abstract
Main text(Cell Reports 25, 1241–1254.e1–e5; October 30, 2018)Figure S4B. Vessel density and tissue morphogenesis were not affected in organs of Apj-CreER;R26-GFP;Kdrfl/fl and Apj-CreER;R26-GFP;Kdrfl+ mice with Tam induction, related to Figure 4 (original)View Large Image Figure ViewerDownload Hi-res image Download (PPT)Figure S6B. Vessel density and tissue morphogenesis were not affected after genetic ablation of Apj+ vessels, and ablation in bulky tumors could also suppress tumor growth, related to Figure 6 (corrected)View Large Image Figure ViewerDownload Hi-res image Download (PPT)Figure S6B. Vessel density and tissue morphogenesis were not affected after genetic ablation of Apj+ vessels, and ablation in bulky tumors could also suppress tumor growth, related to Figure 6 (original)View Large Image Figure ViewerDownload Hi-res image Download (PPT) Main text(Cell Reports 25, 1241–1254.e1–e5; October 30, 2018)Figure S6B. Vessel density and tissue morphogenesis were not affected after genetic ablation of Apj+ vessels, and ablation in bulky tumors could also suppress tumor growth, related to Figure 6 (corrected)View Large Image Figure ViewerDownload Hi-res image Download (PPT)Figure S6B. Vessel density and tissue morphogenesis were not affected after genetic ablation of Apj+ vessels, and ablation in bulky tumors could also suppress tumor growth, related to Figure 6 (original)View Large Image Figure ViewerDownload Hi-res image Download (PPT) (Cell Reports 25, 1241–1254.e1–e5; October 30, 2018) Apj+ Vessels Drive Tumor Growth and Represent a Tractable Therapeutic TargetZhao et al.Cell ReportsOctober 30, 2018In BriefAngiogenesis underpins tumor growth and metastasis, but current targets are suboptimal. Zhao et al. report that Apj is a cell surface marker of pathological angiogenesis and a tractable therapeutic target for tumors. Full-Text PDF Open Access
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