Abstract
We present a case of a patient with Anton's syndrome (i.e., visual anosognosia with confabulations), who developed bilateral occipital lobe infarct. Bilateral occipital brain damage results in blindness, and patients start to confabulate to fill in the missing sensory input. In addition, the patient occasionally becomes agitated and talks to himself, which indicates that, besides Anton's syndrome, he might have had Charles Bonnet syndrome, characterized by both visual loss and hallucinations. Anton syndrome, is not so frequent condition and is most commonly caused by ischemic stroke. In this particular case, the patient had successive bilateral occipital ischemia as a result of massive stenoses of head and neck arteries.
Highlights
Denial of loss of vision, which is associated with confabulation in the setting of obvious visual loss and cortical blindness is known as Anton’s syndrome [1]
Visual anosognosia is frequently believed to represent cortical phenomenon, it is probably more often caused by parietal white matter injury leading to a disconnection syndrome [4, 5]
We present a case of patient with Anton’s syndrome due to bilateral occipital ischemic lesions as a result of massive stenoses of head and neck arteries
Summary
Denial of loss of vision, which is associated with confabulation in the setting of obvious visual loss and cortical blindness is known as Anton’s syndrome [1]. Bilateral occipital brain damage results in blindness; patients start to confabulate to fill in the missing sensory input. Why patients with Anton’s syndrome deny their blindness is unknown, there are many theories. Visual anosognosia is frequently believed to represent cortical phenomenon, it is probably more often caused by parietal white matter injury leading to a disconnection syndrome [4, 5]. We present a case of patient with Anton’s syndrome due to bilateral occipital ischemic lesions as a result of massive stenoses of head and neck arteries
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