Abstract
ISCHEMIA-reperfusion injury in transplantation can cause primary nonfunction and is a matter similar in importance to rejection. Recent studies indicate that neutrophil infiltration plays an important role in ischemiareperfusion injury. We previously reported that there was a correlation between the degrees of mucosal damage and the peak level of IL-8 (CINC), known as a leukocyte chemoattractant, after the reperfusion, and that IL-8 (CINC) is a potential mediator of the ischemia-reperfusion injury in rat small intestine. Thus, we investigated whether anti-rat IL-8 (CINC) monoclonal antibody (mAb IL-8) administration reduces the ischemia-reperfusion injury.
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