Abstract
Aspirin and nonsteroidal antiinflammatory drugs (NSAIDs) have long been thought to provide more than analgesia. For example, compelling data support reduction of colon cancer risk. Although beneficial effects on breast cancer risk have been modest at best, an underlying hypothesis is that aspirin and NSAIDs inhibit cyclooxygenase-2–mediated prostaglandin E2 synthesis, which in turn may lower estrogen levels by inhibiting aromatase activity. If this mechanism is valid, one might …
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Published version (
Free)
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
