Abstract
SummaryHydroxylamine when administered to immobilized cats in acute experiments in doses of 10-20 mg/kg markedly reduced duration and spread of electrically-induced afterdischarges while increasing cerebral levels of γ-aminobutyric acid. These findings increase the probability that a causal relationship may exist between metabolism of γ-aminobutyric acid in a particular cerebral area and its excitability.
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Schedule a callMore From: Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.)
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