Abstract
The strategies implemented to identify pathogenic strains of Salmonella in countries with high production and consumption when is of chicken meat [such as Mexico), successfully bring germ-free meat to the market. Two Salmonella enterica enterica strains obtained from Mexican chicken meat were completely sequenced. The genomic comparison with the CT18 Salmonella strain indicates that strains 103 and 2199 vary by 1.9%. Genome analysis of the isolated strains revealed the presence of numerous virulence genes, as well as antibiotics resistance genes in these two isolates. Their potential pathogenicity was inferred from presence of 22 (103 strains) and 19 genes (2199 strains) homologous to the one annotated in Salmonella enterica virulome databanks. The characterization of these strains will contribute to successful Salmonella monitoring in Mexico.
Highlights
IntroductionThe pathogenicity of a Salmonella strain is related to the presence of distinct genes, such as the avrA gene, only observed in serovars that have a potential to cause severe salmonellosis in humans [4] [5] The vast majority of the world countries have established strict normativity regarding food supplies and regarding Salmonella sp
Their potential pathogenicity was inferred from presence of 22 (103 strains) and 19 genes (2199 strains) homologous to the one annotated in Salmonella enterica virulome databanks
Salmonella enterica enterica food poisoning has been under scrutiny, as the pathogen can cause symptoms ranging from asymptomatic carriers to severe diarrhea [2]
Summary
The pathogenicity of a Salmonella strain is related to the presence of distinct genes, such as the avrA gene, only observed in serovars that have a potential to cause severe salmonellosis in humans [4] [5] The vast majority of the world countries have established strict normativity regarding food supplies and regarding Salmonella sp. Genes related to the severity of Salmonella intoxication have been studied and classified as follow: avrA genes codes for SPI-1 effector protein inhibiting inflammation and apoptosis. The presence of this gene family would favor the Salmonella persistence, slowing the natural clearance of the intestinal cells of the infected patient [2]. When this gene is inverted, flagelin 2 is expressed allowing the microorganism movement [11]
Sign-in/Register to view highlights & summary Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
